OSTEOPOROSIS- INFORMATION!

Written by Dr. Deborah Baker © 2006-2008

With Information from Michael Rae, Researcher Extraordinaire, Advanced Orthomolecular Research
Calgary, Alberta

Calcium is the most abundant mineral in the body. The average adult has about 3 lbs of it in their bones, teeth and blood. Besides building bones, and teeth it is used in vital bodily functions such as maintaining heart beat, transmitting nerve impulses and clotting blood.

It is not just a case of calcium and Vit. D to be considered when thinking about osteoporosis. There are probably about 2 dozen nutrients and hormonal interplays needed to support the matrix of the bone tissue. This is a life-long project. A better bone mass before menses begin in the case of women makes for a much better chance of avoiding osteoporosis in later years.

Get enough calcium. Current "official" recommendations suggest an intake of 1000 milligrams of calcium for younger adults, and 1200 milligrams for people over the age of 50. Some evidence suggests that a still higher intake (1300-1600 milligrams) of calcium is more effective for lowering fracture risk in the elderly. But these numbers are your total calcium need. The more calcium you get in your diet, the less you need from supplements. There is little evidence that ever-higher intake of calcium does your bones any additional good, and indeed taking too much calcium can inhibit the absorption and utilization of other important bone nutrients, such as zinc and copper

Too many health-conscious people believe that conventional calcium supplements (or conventional calcium plus vitamin D) can put an end to bone loss. They can't. As multiple studies have documented, conventional calcium supplements - such as calcium gluconate, calcium citrate, calcium carbonate, and even calcium citrate-malate - slow, but do not halt or reverse, menopausal bone loss, whether taken alone or with vitamin D. Even a total daily calcium intake of 3000 milligrams of calcium from conventional sources isn't enough to stop bone loss, let alone turn the decline around. You simply can't force the bones to take in more calcium, and build more bone, by taking more and more calcium: the mineral itself can only support your existing bone mass, or the building of bone induced by the other factors in your skeletal health program.

But there is one seeming exception. Ossein microcrystalline hydroxyapatite complex (MCHC) consistently halts, or even reverses, bone loss in controlled human trials. When put head-to-head against other calcium supplemental forms, MCHC consistently trumps the conventional calcium supplement. But actually, this is the exception that proves the rule, because MCHC's bone-building powers do not lie in the calcium itself.

True MCHC is not just a form of calcium, but is a calcium-based crystalline nutrient complex, which is how the mineral is actually stored in your bones. Supplements do exist which contain "calcium hydroxyapatite" which lack this crucial nutrient matrix, either because the "calcium hydroxyapatite" is not derived from bone but from chemical synthesis (this is also known "calcium orthophosphate"), or because it uses bone meal, which is heat-treated ("ashed"), breaking down the MCHC crystalline structure and destroying the non-mineral components of the complex. But these supplements, even though they contain the same chemical form of calcium, fail to reproduce the unique effects of MCHC on parameters of bone health.

Thus, the unique support for bone health provided by MCHC is probably due to a combination of its intact crystalline structure, and the vibrant blend of peptides, mucopolysaccharides, and growth factors which accompany the calcium in true MCHC supplements - factors which are not present in conventional calcium supplements, in bone meal, or in synthetic hydroxyapatite. The bottom line is that the effects of MCHC derive from the whole supplement, and not just from its calcium content.

Unfortunately, of course, vegetarians cannot consume MCHC because it is an animal product (although premium MCHC supplements use free-range, pasture-fed livestock from countries like New Zealand or Australia as sources for the raw materials). For vegetarians, the best calcium source is calcium citrate-malate.

Calcium citrate-malate is not the same thing as calcium citrate, or as a simple admixture of calcium citrate and calcium malate. Calcium citrate-malate is prepared in such a way that a significant number of its calcium atoms are bound to both citrate and malate molecules at once. This unique form makes calcium citrate-malate six to nine times more easily dissolved in the stomach than plain calcium citrate.

This superior solubility may be at least part of the reason for the fact that calcium citrate-malate is considerably better-absorbed than calcium citrate. In fact, despite what is often said, nearly all studies have reported that plain calcium citrate is actually no better absorbed than calcium carbonate when taken with food. Most studies find that about 22 to 26% of calcium from calcium carbonate or citrate is absorbed, whereas calcium citrate-malate absorption is consistently found to be around 36 to 37%% in capsules and tablets and can be as high as 42% when dissolved in orange juice.

How Rumors Get Started

The widespread myth of calcium citrate's superior absorption is in part the result of poorly-designed studies, which used calcium excretion as a measure of absorption. The reasoning for using this method is based on the fact that, once your body has used all of the calcium which it can at the time that a dose of calcium is taken in, any extra calcium initially absorbed will then be passed out in the urine. Thus, by giving a dose of calcium so high that the body can't use it all, and then measuring how much calcium passes out through the urine, the comparative bioavailability of two calcium forms can in theory be gauged by seeing how much calcium excretion they cause.

That's a sensible-sounding and inexpensive testing method, and in many cases it probably gives a good picture of calcium absorption. But it falls down in comparing calcium citrate with the carbonate salt. First, the alkalinizing effect of the carbonate reduces the amount of calcium excreted through the urine, making its absorption look lower; and then, some studies suggest, the citric acid in calcium citrate increases the body's excretion of calcium, making its absorption look higher!

Faith in calcium citrate's higher bioavailability was also shored up by a recent "meta-analysis" paper. Meta-analysis is done by combining the results of several separate studies into one mondo-report, which gives a clearer picture of the overall results of the available scientific evidence. But the authors of this meta-analysis made one critical mistake: in combining studies, they assumed that calcium citrate was basically the same as calcium citrate-malate, and lumped the results for the two forms together. In fact, of course, the two forms are considerably different. By combining studies on calcium citrate with studies on the much more bioavailable citrate-malate form, the citrate salt aquired an undeserved glitter, reflected from citrate-malate's radiance.

On the other hand, the hype surrounding so-called "ionic coral calcium" is not the result of understandable errors in otherwise solid science, but of a lack of even the most elementary scientific credibility. Not one clinical trial has ever been performed using this calcium source to show that it is better absorbed or better utilized than other conventional calcium sources. Instead, astoundingly, the claims of high bioavailability for "coral calcium" are not based on controlled studies in humans, but on the stuff's ability to dissolve in water; and as has been shown, such a silly test bears little relationship to the ability of a living body do absorb calcium.34 Indeed, this kitchen-counter method of testing absorption leads to ridiculous exaggerations of calcium absorption, such as 50% absorption for calcium citrate, or 95% absorption for "coral calcium" itself. In the real world, no calcium source has such a high bioavailability.

Calcium citrate-malate has been used successfully in many controlled trials to support bone mass and/or to lower fracture risk. Some of these trials have involved a direct face-off between calcium citrate-malate and other forms of calcium. Such trials demonstrate that, as might be expected from its greater bioavailability, calcium citrate-malate gives better protection to the bones than other vegetarian calcium sources - although its effects are still not as impressive as those of MCHC.

Menatetranone - Vitamin K2

Vitamin K is an essential nutrient, best known for its role in blood clotting. Plants make one form of vitamin K (phylloquinone, or vitamin K1) for their use. But your body doesn't use all of the K1 in your diet "as is." Instead, the body converts some of this plant form of the vitamin into a different vitamin K molecule: Menatetrenone, or MK-4, a form of vitamin K2. Tissues vary in their vitamin K needs, and it's become clear that some tissues have a specific need for Menatetrenone which is not met by phylloquinone. For some purposes (like blood clotting), phylloquinone works fine; but extensive evidence shows that Menatetrenone has unique effects on bone health not shared by phylloquinone.

Fracture victims' levels of Menatetrenone are more depressed than are their levels of phylloquinone.
Areas where more K2 is consumed in the diet have lower fracture rates.
Menatetrenone inhibits the resorption (teardown) of bone caused by the local cellular messenger prostaglandin E2 (PGE2). The same concentration of phylloquinone has no effect. Menatetrenone also cuts down on the bone cells' formation of PGE2 in the first place.
Menatetrenone is able to reduce the creation of osteoclasts (cells involved in the teardown of bone tissue) out of early cell types - but again, phylloquinone has no such power.
Menatetrenone, but not phylloquinone, actually increases the programmed cell death ("apoptosis") of existing osteoclasts.
Menatetrenone strengthens the bone-building legions of the osteoblasts (cells involved in the manufacture of new bone), mildly increasing both their numbers and their activity.

Over the course of the last decade, at least sixteen clinical trials have been performed using Menatetrenone, and every single one has found that K2 supplements protect bone health. Menatetrenone not only slows, halts, or even reverses loss of bone mass: it dramatically reduces your risk of suffering a fracture.

In one trial, women who took an ultra-high dose Menatetrenone supplement for 24 weeks increased their bone mineral density by an impressive 2.2%, even as the women taking a placebo (dummy pill) lost 7.31% of their bone density.

In another trial, Menatetrenone was put to the test in a direct comparison against the bisphosphonate drug etidronate (Didrocal®). Menatetrenone preserved bone mass, and also slashed fracture risk by roughly two thirds over the course of two years.

In a third trial, osteoporotic women taking Menatetrenone supplements sustained nearly no bone loss over two years, while cutting fracture risk by 64% as compared with non-supplementing women.

The ability of bones to withstand fractures is not just determined by the quantity of bone (as measured by Bone Mineral Density (BMD)), but also by the quality of bone - bone "microarchitecture," including especially "trabecular connectivity." Evidence suggests that Menatetrenone's most important effects are on bone quality, not bone quantity.

MENATETRANONE (PEAK K2)...IS NOT LONGER AVAILABLE.

HEALTH CANADA HAS PROHIBITED ITS SALE.

Strontium

Bone loss accelerates suddenly in menopausal women because the drop in estrogen levels causes an increase in the resorption (teardown) of existing bone. But resorption is only half of the story. Age-related bone loss is also caused by a decrease in the formation of new bone tissue.

Existing drugs for treating osteoporosis, as well as calcium and vitamin D supplements, work by reducing bone resorption. But they do not support the formation of new bone. These drugs and nutrients increase the mineralization of bone, but they do not help the body to build new bone tissue. The resulting bone is less prone to fracture, but is not the same as youthful, healthy bone.

Strontium is a mineral found along with calcium in most foods. Research has long suggested that it may be an essential nutrient required for the normal development, structure, function, and health of the skeletal system. Clinical trials going back into the 1940s have supported this conclusion, but recent studies have provided evidence that it can offer unique nutritional support against loss of bone structure and function.

Human clinical trials also support Strontium's ability to both support new bone formation and prevent excessive resorption.

Early clinical trials' results led researchers to speculate that Strontium increased osteoblast activity.
Bone biopsies from a small pilot trial revealed an astounding 172.4% increase in new bone formation after six months of Strontium supplementation.
The bone-building activity of osteoblasts can be measured using bone-specific alkaline phosphatase, while crosslinked N-telopeptide (NTx) and C-telopeptide (CTx) mark the degradation of bone collagen by ravaging osteoclasts. The use of these tests in large clinical trials has confirmed that Strontium supplements decrease bone resorption and also stimulate bone-building osteoblast activity and new bone formation in women with osteoporosis.

Unlike the range of side-effects that accompany bisphosphonates and other antiresorptive drugs, no side-effects have ever been reported that could be attributed to Strontium. People experienced no symptomatic or chemical or physiological signs of toxicity after taking Strontium supplements for as long as four years, at two and a half times the dose of elemental Strontium that's used in today's clinical trials.

TO ORDER STRONTIUM PLEASE CLICK HERE

NEWEST INFORMATION ON STRONTIUM
FOR OSTEOPOROSIS

Not Like “Chocolate in Your Peanut Butter”
High-Dose Strontium and Calcium Don’t Go Well Together

Advanced Orthomolecular Research (AOR) is proud to be an innovator in the nutraceutical world, researching novel, science-backed orthomolecules and bringing them into the hands of health-conscious people and life extensionists first.

Such has been the case with many key supplements, including R(+)-lipoic acid (a world’s first), Benfotiamine (the first in North America), and recently, the bone health powerhouse strontium (AOR’s Strontium Support was the first supplement in the world to deliver the doses used in clinical trials).

But in the time since AOR first released Strontium Support, “me too” supplements have recently begun to appear. These supplements have attempted to make sales by including strontium as part of a “complete” bone health supplement, featuring among other things the old standby, calcium.

Now, calcium is undeniably a key bone health nutrient, and it’s important for users of any strontium supplement to ensure that they are also getting enough calcium. Animal studies suggest that strontium is not effective, and may even be counterproductive, if your calcium intake is not adequate. But including the full dose of strontium in combination with calcium is a sure way to negate most of the benefits of your strontium supplement.

If the formulators of these new knockoff supplements had done their homework, they would have known that high-dose strontium supplements should absolutely not be combined with calcium in one formulation.

The reason for this was made plain in a recent review by Dr. Jean-Yves Reginster, an investigator with the World Health Organization (WHO) Collaborating Center for Public Health Aspects of Rheumatic Diseases, and with the Bone and Cartilage Metabolism Unit of the University of Liège.

Dr. Reginster is the author of fourteen peer-reviewed scientific journal articles on the role of strontium in bone health, and was a principal investigator on three of the largest and best-designed trials. In his review, Dr. Reginster specifically notes that “The simultaneous intake of [strontium] and calcium remarkably reduces the bioavailability of [strontium]. This is probably due to competition at the sites of active absorption. Simultaneous food intake also has a negative influence on the bioavailability of [strontium]”. Based on this critical factor, Dr. Reginster recommends that high-dose strontium should not be taken “concomitantly with a meal or a calcium intake.”1

This fact has long been known, and is the basis for the fact that all of the clinical trials using strontium have carefully ensured that the supplement is taken on an empty stomach, away from calcium in food or in supplements. , , , , , , In the largest and best-designed trials,2,3,4,5 women have taken their strontium first thing in the morning, half an hour to an hour before breakfast, and/or three hours after dinner in the evening; they took their calcium supplements separately, with a meal. This is the protocol they recommend at AOR.

Pills or powders which combine calcium with strontium are, therefore, not the “convenient.” “inexpensive” deals they initially seem, but are ill-designed and likely ineffective “kitchen sink” hodgepodges.

Some of these strontium-calcium products then further shoot their users in the foot by using poor forms of key ingredients. Some, for instance, use poor forms of calcium, such as cheap calcium carbonate (which has low bioavailability and which reduces your absorption of other nutrients by neutralizing stomach acid) and synthetic calcium hydroxyapatite (an extremely poorly-absorbed synthetic calcium phosphate salt not to be confused with ossein microcrystalline hydroxyapatite complex (MCHC)).

Others use magnesium carbonate as a magnesium source; this is another antacid, and like calcium carbonate is poorly absorbed.

Likewise, one of these products is even trading off of the research on Menatetrenone (MK-4) – the mammalian form of vitamin K2 and the one used in all of the “vitamin K2” clinical trials – to sell another “vitamin K2:” the unproven, bacterial menaquinones.

It’s a different thing if there is only a small amount of strontium in a core bone health supplement, such as 500 micrograms to 5 milligrams – doses in the range of human dietary intakes. Such doses are appropriate, as they preserve the ratio of calcium and strontium present naturally in whole-food diets. (Such as Ortho-Bone)

In fact, all natural calcium sources also have a small amount of strontium in them, because of the similar metabolism of the two nutrients in living beings.

The presence of calcium with no strontium in calcium supplements might be expected to upset this natural balance, leading to supression of whatever strontium is in your diet and ultimately perturb the natural balance of minerals in your bone.

Indeed, some evidence already exists that, over a lifetime, these low, nutritional doses of strontium do have a role to play in your health. For example, it was discovered in the 1960s that areas with more strontium in the water have a lower incidence of dental caries , – a finding which was to be reinforced by the findings of at least eight more studies over the course of the next few decades.

Everyone concerned about their bone health needs a core calcium supplement, along with other key nutrients such as magnesium, vitamin D3, and Menatetrenone. In such a supplement, a small, nutritional dose of strontium is a good balancing act, reflecting the trace levels of strontium naturally present in food.

But if you need the potent support of a “megadose” strontium supplement, it should absolutely not come in a combination with calcium. Feed your bones these two great “tastes” – but remember, they don’t “taste great together.”

On the heels of this information, we at Y2K Health and Detox Centre are suggesting Strontium Support to those who have been diagnosed with osteoporosis or are susceptible either because of body type or family history to take 2 about 30 minutes before breakfast and 2 in the evening..about 2 hours away from dinner.

Use Ortho-Bone 3-6 at breakfast and 3-6 at dinner. Vitamin Peak K2 can then be taken 1 at breakfast and 1 at dinner.

Stomach Acid:

Calcium must be ‘ionized’ for absorption by the stomach acid naturally present in the gastric environment. The problem is that about 40% of adults at midlife are low in gastric acid. Calcium carbonate is the most common form of calcium supplement and it is the hardest for the stomach to ionize even when normal levels of acid are present. Therefore studies show that at that time of life and later only about 2% of calcium carbonate is absorbed.

Vitamin D: **UPDATED INFORMATION**

Take Enough Vitamin D. Aside from improving calcium absorption, vitamin D is needed for proper muscle function, which may play a role in protecting against fractures. So getting enough vitamin D is important. And you simply can't rely on the sun to meet your requirements, especially in Northern climates. Flat-out vitamin D deficiency is found in one third of otherwise-healthy Canadians at least once over the course of the year.

More recent studies have indicated that up to 70% of adolescents and adults have Vit D levels lower than 40 ng/ml...and anything below 35 ng distinctly reduces calcium absorption from the intestines.

Cardiovascular problems occur more in the winter and at higher and lower altitudes indicating that Vit D has a protective function here. People with levels of 25ng or more over the 36ng level had half as much chance of having heart attacks.

Other health benefits of higher Vit D levels in other studies were:

-better maintained and lower blood glucose levels and hence insulin response..lowering one's chance of developing adult onset diabetes.

-80% lower chance of developing colon cancer.

-much lower rates of knee and hip arthritis.

-the lowest rate of periodontal disease occurs in people with Vit D levels over 40 ng/ml.

-nursing mothers need 4,000 IU of Vit D per day.....NOT the recommended 400 IU. Its no wonder science tells us that most mother's milk is low in Vit D....the mothers are!

Indeed, the whole reason that our milk is now fortified with vitamin D is that rickets (bone disease caused by vitamin D deficiency) was epidemic in children in the Northern United States at the turn of the twentieth century - when kids spent a lot more time out-of-doors than do today's adults. There's a good reason for this: studies in human skin suggest that the amount of sun to which a person in Boston or Edmonton is exposed in the winter is not enough to make the body produce the vitamin. But even in sunny Spain, researchers have found that 80% of children have inadequate vitamin D levels in March and October, and the situation is much the same in France.

From what we now know, the old RDA of 400 IU will not protect you from vitamin D insuffciency except in the sunniest of climates. A controlled trial in teen and preteen girls in Finland showed that a 400 IU vitamin D supplement was not enough to keep serum levels of the active vitamin above the cutoff for insufficiency, and studies in the health of large populations confirm the finding in Canadian and Danish women lead to the same conclusion. More importantly, the use of standard 400 IU supplements have not been shown to reduce fracture rates, , and even 600 IU has little effect on BMD.

So how much vitamin D do you need? For optimal bone health - as opposed to simply avoiding a case of obvious rickets - scientists are now suggesting that the proper test is to see how much of the vitamin it takes to minimize the elevation of parathyroid hormone, which as we've noted leeches calcium from the bones when serum calcium levels are low. To reach this target, doses of as much as 4000 IU per day are recommended by some legitimate authorities,63 and such doses have been used successfully and with apparent short-term safety to achieve the goal.

But it's premature to start using this high a dose: for one thing, taking this much vitamin D may be toxic when taken in the vegetarian form of ergocalciferol (vitamin D2) - although apparently not when you use cholecalciferol (vitamin D3 - the animal-sourced form).63 But clinical trials show that we don't need to self-experiment with these massive doses to get results. Controlled studies show that vitamin D, together with calcium, helps to reduce the risk of fracture at a dose of 800 IU per day.

There is a very intricate process in place in your body for the conversion and production of Vitamin D to occur in your skin, liver and kidney

Skin + Sunlight Þ 7-Dehydrocholesterol Þ D3 (cholecalciferol).

D3 Þ LiverÞ Converted to 25-Hydroxycholecalciferol (which is 5x more potent than D3)

25-HDCC Þ Kidney Þ Converted to 1,25-Dihydroxycholecalciferol (which is 10x more effective as D3).

Buy Vitamin D3 - 5000 mg.....Here

Now we look at a dynamic which rarely looked at when assessing the osteoporotic patient. Do they have any kidney or liver dysfunction. This could include things such as nephritis, being on dialysis, mercury toxic load in the kidney and/or liver, hepatitis, cirrhosis, fatty degenerative liver, toxic overload of any type, chronic medication ingestion, etc. If either or both of these organs are compromised, then so is the enhancement of Vitamin D and its metabolites and its ability to assist in calcium metabolism.

Calcium is controlled by a hormone called parathyroid hormone from four small glands on the corners of the thyroid gland. If calcium levels in the blood become low, then parathyroid hormone is secreted causing releasing of calcium from the bones. Conversely, if calcium levels in the blood are high, parathyroid hormone is inhibited and calcium will be used for bone, building nerve transmission etc. as needed.

The role estrogen plays in bone mass is that as it lowers in life, it makes the osteoclasts, the cells which are responsible for bone breaking down, more sensitive to parathyroid hormone which causes increase breakdown of bones. This results in an increase in circulating calcium for other needs than bone maintenance. Osteoblasts, on the other hand are the cells responsible for building of bone...these are the ones we want to stimulate, particularly in later life to keep bone mineralized and strong. Some of the things we can do follow:

Lifestyle Factors:

Avoid coffee, alcohol and smoking.
Exercise is probably the most important factor of bone mass. Walking and resisted weights are great. Research has shown that this more important than hormone or dietary factors. Even just l hour, 3X per week stops bone loss and actually increases bone mass.
Diet - avoid soft drinks and excess meat. Vegetarians lose less bone. High protein diets have high levels of phosphates which is associated with high excretion of urinary calcium Raising protein from 47 to 142 gm doubles calcium excretion.
Sugar increases calcium excretion (Average consumption is 150 lbs.per year..per adult)
Phosphates in soft drinks especially Coke and Pepsi (it seems the caffeine makes the phosphate problem even more severe) where they are needed to dissolve the sugar and contribute to the taste. (Average is three quarts per week per person)
Good, non dairy sources are dark green leafy vegetables such as kale, collards, parsley, broccoli, althouth research now shows that skim milk is the best source of calcium and goat's milk is better because of the size of the molecules. The proteins and fat particles are very similar in size as human breast milk, accounting for the fact most people who have dairy intolerances, can consume goat milk products..
Other Minerals and Factors:

Magnesium:

Take a Magnesium You Can Absorb. Magnesium is central to various aspects of bone metabolism, and borderline magnesium deficiency is surprisingly common. Unfortunately, far too many bone health formulas rely on magnesium oxide as the source of this mineral, for the simple reason that it takes up less room in a capsule, and therefore requires fewer capsules to be taken to reach the daily dose. But compared to other sources of the mineral, magnesium oxide has "extremely low" bioavailability (22.8%). Additionally, magnesium oxide is an antacid, which can impair digestion and nutrient absorption. This is an especial concern in many older people, whose low stomach acid may even trigger pernicious anemia (flat-out B12 deficiency).

Magnesium citrate is certainly somewhat better, at 29.64% absorption,67 but it's still far from the best magnesium you can choose; and, indeed, much of the supposed "magnesium citrate" on health-food store shelves is not true, fully-reacted magnesium citrate, but a mixture of magnesium oxide and magnesium citrate.

Much better absorption is available from other forms of magnesium. Among the available forms of magnesium, fully-reacted magnesium monoaspartate stands out as the best, with a remarkable 41.7% bioavailability.

Remember the neglected nutrients. Calcium, magnesium, and vitamin D are very well-known as nutrients with an important place in bone health. By contrast, you may never have heard of the powerful support that Menatetrenone and Strontium can lend your bones before reading this special issue of Advances. But there are a host of nutrients important to bone health which are too often neglected in putting together a total lifestyle program. These would most prominently include manganese, zinc, and copper, and would extend to other, even more commonly-neglected nutrients such as silicon, boron, and vitamin C.68 Methylating nutrients such as vitamin B12 and folic acid may also be important to bone health, perhaps because of the toxic effects of homocysteine on the protein fibers in bone.

Boron - 3mg per day decreases urinary excretion by 47% and increases levels of 17-B-estradiol - the most biologically active estrogen, naturally.

Boron also activates Vit D in the kidney.

We would get enough if we ate 3 vegetable servings and 2 fruits per day (best source of boron) and yet recent surveys show that only 51% of people eat one serving.

Do not take all of your calcium at once. Someone who is taking 800mg. per day - if they take it all at once, the absorption rate is about 15%. If it is divided over the day, the absorption can be raised to 40%. The standard of suggesting one consumes 1,000 me per day is based on the fact that 40% is about the max one actually absorbs.

Do not use oyster shell, dolomite or bone meal. In 1981 the FDA warned against these forms because of lead content. One brand had as much as 25 mcg/800 mg and 6mcg/800mg or over is toxic for children.

Vit B6, Folic Acid and Vit B12 - a depletion of these elements results in an increase in homocysteine which leads to a defective bone matrix or structure.

TO ORDER THE CALCIUM/MAGNESIUM SUPPLEMENT I USE FOR MY

PATIENTS..CLICK HERE!

NOW Available - Ortho-Bone The only all inclusive bone support supplement with Strontium and K2 in its formula CLICK HERE

If you would like to ask Dr. Deb. any questions or receive any further information about the professional products found on this site and in our stores, please e-mail to Dr. Deb

Thank You
Dr. Deborah Baker

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